Ciba Foundation Symposium 38 - Lung Liquids by CIBA Foundation Staff

By CIBA Foundation Staff

Chapter 1 creation: pursuits of the assembly (pages 1–2): C. J. Dickinson
Chapter 2 Ultrastructural foundation for Alveolar?Capillary Permeability to Protein (pages 3–28): Eveline E. Schneeberger
Chapter three Permeability of Pulmonary Vascular Endothelium (pages 29–48): A. P. Fishman and G. G. Pietra
Chapter four The Permeability of Lung Capillary and Alveolar partitions as Determinants of Liquid hobbies within the Lung (pages 49–64): L. B. Strang
Chapter five Interstitial Fluid and Transcapillary Fluid stability within the Lung (pages 65–76): Bjarne A. Waaler and Petter Aarseth
Chapter 6 Dynamics of Subatmospheric strain within the Pulmonary Interstitial Fluid (pages 77–100): Arthur C. Guyton, Aubrey E. Taylor, Robert E. Drake and James C. Parker
Chapter 7 impression of Lung Inflation on Alveolar Permeability to Solutes (pages 101–124): Edmund A. Egan
Chapter eight Comparative points of Salt and Water shipping throughout Lung (pages 125–132): R. D. Keynes
Chapter nine delivery of Ions and Water around the Epithelium of Fish Gills (pages 133–159): J. Maetz
Chapter 10 Coupling of Water to Solute move in remoted Gastric Mucosa (pages 161–177): R. P. Durbin
Chapter eleven Ion shipping throughout Amphibian Lung (pages 179–197): John T. Gatzy
Chapter 12 Ion shipping and Water movement within the Mammalian Lung (pages 199–220): R. E. Olver
Chapter thirteen Carbonate Dehydratase (Carbonic Anhydrase) and the Fetal Lung (pages 221–234): T. M. Adamson and B. P. Waxman
Chapter 14 Lung Carbonate Dehydratase (Carbonic anhydrase), CO2 shops and CO2 shipping (pages 235–254): Leon E. Farhi, John L. Plewes and Albert J. Olszowka
Chapter 15 Mechanism of Alveolar Flooding in Acute Pulmonary Oedema (pages 255–272): Norman C. Staub, Marlys Gee and Carol Vreim
Chapter sixteen Intracellular and Subcellular Oedema and Dehydration (pages 273–300): Eugene D. Robin and James Theodore
Chapter 17 scientific issues of Lung Liquid (pages 301–317): C. J. Dickinson
Chapter 18 ultimate feedback (pages 319–321): C. J. Dickinson

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LASSEN,N. A. ) (1970) Capillary Permeability (Alfred Benzon Syrnp. 2), Academic Press, New York ERDMANN, A. , 111, VAUGHAN, T. R. JR, BRICHAM,K . , WOOLVERTON, W. C. & STAUB, N. C. (1975) Circ. Res. 37, 271-285 FISHMAN, J . A. & PIETRA, G . G. (1973) Bull. Mt. Biol. Lab. 13, 36-38 GAAR,K.

The fact that alveolar junctions are tighter than endothelial junctions is entirely consistent with clinical experience. But the suggestion that alveolar junctions must be disrupted in order to open is puzzling. Schneeberger: Yes, we don’t know what factors cause these junctions to come apart, if they come apart at all. Olver: In the lamb fetus the epithelial junctions may open up as lung liquid is absorbed, but Dr Egan will tell us about that later (this volume, pp. 101-1 10). How complex are the tight junctions in the alveolar epithelium in relation to those in other epithelia?

Dickinson: Have you done any comparable morphological investigations on the lung in pulmonary oedema, or are the freezing problems insuperable? Schneeberger: I have not done that, but we would of course like to study the freeze-fracture image of lungs fixed under increased intravascular pressure. In lungs perfused with EDTA or under increased pressure (Hovig et al. 1971) no widening of endothelial junctions was detected. Whether there is widening of the discontinuities i n the strands seen by freeze-fracture has yet to be determined.

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