Global Dimensions of Childhood Obesity by Richard K. Flamenbaum

By Richard K. Flamenbaum

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2] Amigo, H; Bustos, P; Leone, C; Radrigan, ME. Growth deficits in Chilean school children. American Society for Nutritional Sciences, 2000 1, 251-254. Childhood Obesity in Transitional Countries: A South African Perspective 35 [3] Aristimuno, GG; Foster, TA; Voors, AW; Srinivasan, SR; Berenson, GS. Influence of persistent obesity in children on cardiovascular risk factors. Circulation, 1984 69, 895-904. [4] Asayama, K; Hayashi, K; Hayashibe, H; Uchida, N; Kodera, K; Nakazawa, S.

Also a randomized, double-blind, placebo-controlled trial carefully studied the intake of calcium-enriched foods and bone mass growth in pre-pubertal girls. The results suggest a possible positive effect of calcium supplementation on skeletal growth in pre-pubertal girls (see [18]). 6 %) (see [113]). About half (51 %) of the stunted girls and 43 % of the stunted boys came from the rural areas. Other research set within the urban environments of developing countries has suggested that stunting in infancy may increase the future risk of greater fatness and overweight in later childhood (see, for instance, [13]).

Petersen sity remains largely undefined. It has been postulated that nutritional stunting is associated with impaired fat oxidation (see [77]) which could predispose stunted children to excessive weight and fat gains at excessive energy and fat intakes. This was evident in another study where nutritionally stunted children had measurably impaired fat oxidation compared with non-stunted control children living in the same environment. Specifically, stunted children had significantly higher respiratory quotients (RQ) and lower fat oxidation in the fasting state and 30 minutes after a meal (subsequent postprandial meals remained higher but not significantly so).

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