Hypoxia and the Circulation by Martin Burtscher (auth.), Robert C. Roach Ph.D., Peter D.

By Martin Burtscher (auth.), Robert C. Roach Ph.D., Peter D. Wagner, Peter H. Hackett (eds.)

The hypoxia volumes will specialise in leading edge study on the interface of hypoxia and biomedicine. Hypoxia is a continuing hazard to the human physique and its very important organs all through lifestyles. there are lots of occasions during which the possibility is heightened in future health and sickness, yet mechanisms have advanced to reduce its unsafe results.

The overseas Hypoxia Symposia was once based to permit scientists, clinicians, physiologists, immunologists, mountaineers and different contributors to percentage their reviews of the occasions linked to oxygen lack and the variations that permit us to outlive.

The undertaking of the overseas Hypoxia Symposia is to give leading edge, refined learn on the very maximum degrees into the various results of hypoxia on people and animals in future health and disease.

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Huez S, Retailleau K, Unger P, Pavelescu A, Vachiery JL, Derumeaux G and Naeije R. Right and left ventricular adaptation to hypoxia: a tissue Doppler imaging study. Am J Physiol Heart Circ Physiol 289: H1391-H1398, 2005. 30. Kihara Y, Grossman W and Morgan JP. Direct measurement of changes in intracellular calcium transients during hypoxia, ischemia, and reperfusion of the intact mammalian heart. Circ Res 65: 1029-1044, 1989. 31. Kingma I, Tyberg JV and Smith ER. Effects of diastolic transseptal pressure gradient on ventricular septal position and motion.

36. Louie EK, Lin SS, Reynertson SI, Brundage BH, Levitsky S and Rich S. Pressure and volume loading of the right ventricle have opposite effects on left ventricular ejection fraction. Circulation 92: 819-824, 1995. 37. Maughan WL, Kallman CH and Shoukas A. The effect of right ventricular filling on the pressure-volume relationship of ejecting canine left ventricle. Circ Res 49: 382388, 1981. 38. Miller AT, Jr. and Hale DM. Increased vascularity of brain, heart, and skeletal muscle of polycythemic rats.

Visceral adiposity is also the major risk factor for the development of Sleep Apnea (SA) - an association that has fueled interest in the co-morbidity of SA and the metabolic syndrome, but hampered attempts to ascribe an independent causative role for Sleep Apnea in the development of insulin resistance and type 2 diabetes. Numerous population and clinic-based epidemiologic studies have shown associations, often independent of obesity, between SA (or surrogates such as snoring) and measures of glucose dysregulation or type 2 diabetes.

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