Pathogenesis of Bacterial Infections in Animals, Third

This much-anticipated 3rd version back consolidates the information of greater than twenty specialists on pathogenesis of animal disorder attributable to a variety of species or teams of micro organism. Emphasizing pathogenic occasions on the molecular and mobile degrees, the editors and individuals position those advancements within the context of the general photo of illness. Pathogenesis of Bacterial Infections in Animals, 3rd edition, updates and expands the content material of the second one variation and comprises state-of-the-art details from the most up-tp-date research.

Comments on earlier editions:

"...highly recommended."
--The Veterinary Record

"...a accomplished, entire and easy-to-use resource of information."
--Veterinary Microbiology

"...recommended for graduate scholars and experts in microbiology, pathology and infectious disease."
--U.S. Animal overall healthiness organization Newsletter

"...a fantastic book."
--Journal of the yank Veterinary scientific Association

"...highly recommended."
--The Cornell Veterinarian

Graduate scholars, school, researchers, and experts in microbiology, pathology, and infectious illnesses will take advantage of this highly-detailed and elevated version of a well-liked and well-read veterinary textual content.

Content:
Chapter 1 issues in Bacterial Pathogenic Mechanisms (pages 3–12): C. L. Gyles and J. F. Prescott
Chapter 2 Evolution of Bacterial Virulence (pages 13–22): P. Boerlin
Chapter three Streptococcus (pages 23–42): J. F. Timoney
Chapter four Staphylococcus (pages 43–55): okay. Hermans, L. A. Devriese and F. Haesebrouck
Chapter five Bacillus Anthracis (pages 57–67): M. A. Weiner and T. C. Dixon
Chapter 6 Mycobacterium (pages 69–76): C. O. Thoen and R. G. Barletta
Chapter 7 Corynebacterium and Arcanobacterium (pages 77–86): B. H. Jost and S. J. Billington
Chapter eight Rhodococcus (pages 87–98): J. F. Prescott, J. Ren and C. Dupont
Chapter nine Listeria (pages 99–110): C. Czuprynski
Chapter 10 Erysipelothrix Rhusiopathiae (pages 111–116): Y. Shimoji
Chapter eleven Neurotoxigenic Clostridia (pages 117–124): R. H. Whitlock
Chapter 12 Histotoxic Clostridia (pages 125–130): J. G. Songer
Chapter thirteen Enteric Clostridia (pages 131–142): J. G. Songer
Chapter 14 Salmonella (pages 143–167): S. J. Libby, T. A. Halsey, C. Altier, J. Potter and C. L. Gyles
Chapter 15 E. Coli Shigella (pages 169–191): T. Adam and C. L. Gyles
Chapter sixteen Escherichia Coli (pages 193–223): C. L. Gyles and J. M. Fairbrother
Chapter 17 Actinobacillus (pages 225–241): J. I. MacInnes and J. T. Bosse
Chapter 18 Haemophilus (pages 243–257): T. J. Inzana and L. Corbeil
Chapter 19 Bordetella (pages 259–272): D. A. Bemis and B. Fenwick
Chapter 20 Pasteurella and Mannheimia (pages 273–294): J. D. Boyce, R. Y. C. Lo, I. Wilkie and B. Adler
Chapter 21 Yersinia (pages 295–307): J. Mecsas and R. Chafel
Chapter 22 Brucella (pages 309–319): S. C. Olsen, C. O. Thoen and N. F. Cheville
Chapter 23 Pseudomonas (pages 321–342): M. Matewish and J. S. Lam
Chapter 24 Moraxella (pages 343–352): W. P. Michalski and J. L. Farn
Chapter 25 Campylobacter and Helicobacter (pages 353–361): L. A. Joens
Chapter 26 Lawsonia Intracellularis (pages 363–372): C. J. Gebhart and R. M. C. Guedes
Chapter 27 Gram?Negative Anaerobes (pages 373–383): D. J. Hampson, T. G. Nagaraja and N. B. Buller
Chapter 28 Leptospira (pages 385–396): B. Adler and A. de l. a. Pena?Moctezuma
Chapter 29 Mycoplasma (pages 397–414): ok. L. Whithear and G. F. Browning
Chapter 30 Chlamydia (pages 415–424): A. A. Andersen
Chapter 31 Rickettsiales (pages 425–444): S. Harrus, T. Waner, S. Mahan and H. Bark

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Additional info for Pathogenesis of Bacterial Infections in Animals, Third Edition

Sample text

It is the most frequently isolated pathogen from equine joints, lymph nodes, nasal cavities, lungs (Hoffman et al. 1993), and uterus. In situations of concurrent influenza virus infection, high summer temperature, or transport stress, it can be a devastating and rapidly fatal pathogen in the respiratory tract. S. zooepidemicus is also a serious zoonotic pathogen of humans who may become infected by exposure to contaminated milk products or by contact with horses. VIRULENCE FACTORS S. zooepidemicus produces many of the virulence factors described for S.

Evers (1968) reported bacteremia in horses inoculated intranasally with S. equi and in noninoculated contact horses that became infected. Blood cultures were more likely to be positive on days 6 to 12 following inoculation. 3. These streptococci show long chains and large numbers of degenerating PMNs typical of extracellular growth in the absence of opsonizing antibodies. not been confirmed but clearly show the potential for localization of S. equi in body sites other than the lymph nodes of the head and neck and for the formation of circulating immune complexes.

Equi infection until weaning. The antiphagocytic SeM is widely believed to be a major protective antigen of S. equi. This heat- and 31 acid-resistant protein antigen is protective for mice (Timoney and Trachman 1985) and is an important component of commercial strangles vaccines. However, a SeM-negative mutant of S. equi protected horses against experimental challenge following intranasal vaccination (Timoney et al. 2000). Thus, antigens other than SeM are involved in protective responses. Vaccines containing either the acid-extracted or enzymatically extracted M protein are very effective in stimulating serum opsonic bactericidal antibodies when administered in a course of two inoculations, but have given a disappointing performance under conditions of natural exposure.

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