Transgenic Herbicide Resistance in Plants by V. S. Rao

By V. S. Rao

This booklet offers a entire and in-depth dialogue at the improvement of herbicide resistance prior to now 50 years, emphasizing the biochemical pathways of herbicide resistance in weeds. It discusses the rules of plant genetics, assorted tools of genetic engineering, makingof transgenic crops, a variety of transgenic vegetation conferred with herbicide resistance, evolution of weed, difficulties next to becoming of transgenic vegetation, merits and dangers of growing to be transgenic vegetation, and administration of transgenic plants. jam-packed with updated details, the booklet comprises proper references, facts, figures, and illustrations.

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Extra resources for Transgenic Herbicide Resistance in Plants

Example text

One of them is that stacking of multiple genes in one germplasm takes many years, but still faces the possibility of segregation of transgenes in later generations [Yu and Birchler 2007; Yu et al. 2007; Xu et al. 2012]. Another limitation is linkage drag, which refers to the reduction in fitness of a cultivar due to deleterious genes introduced along with the beneficial gene during backcrossing [Xu et al. 2012; Yu and Birchler 2007]. A mini-chromosome is an extremely small version of a chromosome, the threadlike linear strand of DNA and associated proteins that carry genes and functions in the transmission of hereditary information.

Genes, with each one causing a small increase in fitness under 24 Transgenic Herbicide Resistance in Plants herbicide selection, may systematically promote increased fitness in genotypes as genetic recombination occurs over successive generations. Implicitly, the rate of evolution is likely to be slower than for single nuclear-encoded genes. Awareness of the possibility of quantitative resistance has led to recommendations to apply labeled herbicide rates to weeds of the size recommended by the registration.

Thus, the enzyme spared by the herbicide will carry on the normal plant metabolic activities. 7. Modification of cell membrane function and structure. 8. Altered sensitivity of the key target enzyme caused by mutation(s). 9. Enhanced metabolic breakdown and conjugation of the herbicide. 10. Enhanced degradation of herbicide-generated toxic products. These mechanisms, and consequently the expression of resistance, are controlled by genetic loci. Inheritance of Herbicide Resistance There are three modes of inheritance of herbicide resistance: Nuclear inheritance, Cytoplasmic inheritance, and Quantitative inheritance.

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